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Issue Brief from the Ornithological Council

Volume 1, Number 2

August 1997
 

MERCURY CONTAMINATION IN BIRDS AND HUMANS

 

THE ISSUE

POLICY ISSUES

BACKGROUND

Mercury in lakes and rivers originates from industry (coal burning operations, smelters, incineration of waste) and the inappropriate disposal of millions of mercury batteries. Coal industries in the U. S. release approximately 100 tons of mercury into the atmosphere each year and 3-5,000 tons are released worldwide. Once released into the air, mercury can be transported thousands of miles. When deposited, metals do not degrade in the environment and levels are rising each year. In the water, inorganic mercury is chemically altered and transformed into more toxic methylmercury. Methylmercury becomes more concentrated at each level up the food chain and has serious ramifications for animals at high levels in the food chain such as fish-eating birds and humans.

THE FACTS

An EPA report (1995) explains that areas highest in mercury contamination are the Northeast, Great Lakes, Florida, and the West. Because of the release of mercury by industries all over the world and mercury's mobility in the atmosphere, mercury emissions are an international problem. In any case, we still need research to determine the sources of mercury released into the atmosphere and to provide a biologically sound the basis for legislation regulating mercury emissions.

 As a precautionary measure, the U.S. Food and Drug Administration discourages human consumption of fish containing mercury levels greater than or equal to 1.0 part per million (ppm). Consumption of mercury-contaminated fish by birds, such as eagles, and herons, results in the accumulation of methylmercury in their tissues where it continues to build up over the life of the bird. Neurological damage and reproductive failure from mercury poisoning have been found in birds. Bald eagle eggs have been killed by mercury concentrations over 1.0 ppm. Concentrations as high as 19.1 ppm have been found in feathers of eagle chicks and cause sterility. In other cases adult birds have been killed by high concentrations, eggs fail to hatch, flight is impaired, aberrant behavior and kidney lesions develop.

The adverse effects of mercury contamination - neurological impairment, decreased reproductive success, and physiological impairment in birds and humans - may go unnoticed for months and even years. Until research can be conducted to document the sources of mercury we cannot write supporting legislation to reduce levels. Children, pregnant women and sensitive species of wildlife, such as fish-eating birds, continue to remain at risk of mercury contamination.

Further Reading:

Furness, R. W. & J.J.D. Greenwood. 1993. Birds as monitors of pollutants. In "Birds as Monitors of Environmental Change. Chapman & Hall, New York.

 Welch, L. 1994. Contaminant burdens and reproductive rates of bald eagles breeding in Maine. M.S. thesis, Univ. of Maine, Orono, ME.

 Zillioux, E.J., D.B. Porcella, & J.M.Benoit. 1993. Mercury cycling and effects in freshwater Wetlands ecosystems. Environ. Toxicology & Chemistry 12: 1-120.

This publication has been reviewed by professional ornithologists under the auspices of the Ornithological Council. For further information you may contact the Ornithological Council. Produced with the assistance of the Office of Migratory Bird Management, US Fish and Wildlife Service.

 Citation: Ornithological Council (1997). Mercury Contamination in Birds and Humans. Bird Issue Brief Vol.1, No. 2.


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